研究
2017 研究業績・論文発表
論文
Gao Y, Wake H, Morioka Y, Liu K, Teshigawara K, Shibuya M, Zhou J, Mori S, Takahashi H, Nishibori M.
Phagocytosis of Advanced Glycation End Products (AGEs) in Macrophages Induces Cell Apoptosis.
Oxid Med Cell Longev. 2017;2017:8419035.
マクロファージによるAGE貪食によるアポトーシス誘導効果についての検討
Watanabe M, Toyomura T, Wake H, Liu K, Teshigawara K, Takahashi H, Nishibori M, Mori S.
Advanced glycation end products attenuate the function of tumor necrosis factor-like weak inducer of apoptosis to regulate the inflammatory response.
Mol Cell Biochem. 2017 Oct;434(1-2):153-162.
AGE/TNF-αによるアポトーシス誘導効果についての検討
Morioka Y, Teshigawara K, Tomono Y, Wang D, Izushi Y, Wake H, Liu K, Takahashi HK, Mori S, Nishibori M.
The specific localization of advanced glycation end-products (AGEs) in rat pancreatic islets.
J Pharmacol Sci. 2017 Aug;134(4):218-224.
糖尿病の障害メディエーターのAGEsのラ氏島細胞障害機序の検討
Fu L, Liu K, Wake H, Teshigawara K, Yoshino T, Takahashi H, Mori S, Nishibori M.
Therapeutic effects of anti-HMGB1 monoclonal antibody on pilocarpine-induced status epilepticus in mice.
Sci Rep. 2017 Apr 26;7(1):1179.
障害メディエーターHMGB1のてんかん発作への関与の検討
Wang D, Liu K, Wake H, Teshigawara K, Mori S, Nishibori M.
Anti-high mobility group box-1 (HMGB1) antibody inhibits hemorrhage-induced brain injury and improved neurological deficits in rats.
Sci Rep. 2017 Apr 10;7:46243.
障害メディエーターHMGB1の出血性脳障害への関与の検討